Who Can Benefit?
People with Multiple Sclerosis and other diagnoses that lead to motor disabilities, sensory disorders and functional problems. Mollii has also been effective in reducing acute and chronic pain, alleviating mood disorders and calming anxieties, providing practitioners and clients with a new, complementary tool to existing therapies. Mollii is also proving to have positive effects on physical performance at an elite level when applied before activity and on recovery.
- Exercise Physiologists
- Social workers
- Case managers
Health professionals are encouraged to write a referral, however a referral is not necessary for clients to book an appointment. Health professionals are welcome to attend Mollii sessions, as long as their client gives consent, and where is has been previously arranged with the clinic.
Métier Medical is a Registered NDIS Provider, working closely with the NDIS in funding the Mollii Suit.
Mechanism of Action?
The Mollii method uses low frequencies and low intensities that evokes sensory input but does not directly elicit muscle contractions. The principle mechanism in the Mollii approach is the activation of the disynaptic reciprocal Ia inhibitory pathway. Relaxation of the agonist muscle is achieved by the electrical stimulation of the antagonist (opposite) muscle, thus enhancing contraction of the agonist and voluntary movements.
Treatment with Mollii may be relevant for focal, segmental and general dystonia although controlled clinical trials are needed. Clinical experiences suggest that Mollii may reduce dystonic symptoms and maintain body posture. The sensory input provided by both electrical stimulation and the physical sensation of the tightly fitted suit may also have an impact on proprioceptive awareness, which is essential not only for motor control in dynamic activity but also for maintaining position and balance control.
Mollii may reduce pain through both central and peripheral mechanisms. In the central nervous system, the activation of opioid, GABA, serotonin, and muscarinic receptors, induced by Mollii, may reduce pain related dorsal horn neuron activity. In peripheral nerves, opioid and α-2 noradrenergic receptors are involved in electrical stimulation-induced analgesia at the site of stimulation.
Low-frequency ES electrical stimulation activates μ-opioid receptors in the spinal cord and the brainstem leading to a decreased sensation of pain. Spinal serotonin concentrations are also increased during and immediately after treatment.